ferrostatin 1 Search Results


ferrostatin 1  (MedChemExpress)
99
MedChemExpress ferrostatin 1
Ferrostatin 1, supplied by MedChemExpress, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ferrostatin 1  (Selleck Chemicals)
96
Selleck Chemicals ferrostatin 1
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ferrostatin  (Santa Cruz Biotechnology)
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Santa Cruz Biotechnology ferrostatin
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lethal 3 m6a n6 methyladenosine w writers e erasers r readers kp krasg12d  (Cell Signaling Technology Inc)
94
Cell Signaling Technology Inc lethal 3 m6a n6 methyladenosine w writers e erasers r readers kp krasg12d
Fig. 8. Schematic presentation of the study. Briefly, the <t>m6A</t> reader YTHDC2 is an endogenous ferroptosis inducer and its function is to suppress system XC −. In addition to what we’ve previously reported that the mRNA stability of SLC7A11 is suppressed by YTHDC2, in the present study we also found that SLC3A2, another subunit of system XC − can also be suppressed by YTHDC2 via targeting HOXA13-mediated transcription of SLC3A2, and the m6A methylation and subsequent destabilization of HOXA13 mRNA is a prerequisite for this process.
Lethal 3 M6a N6 Methyladenosine W Writers E Erasers R Readers Kp Krasg12d, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ferrostatin 1  (Tocris)
92
Tocris ferrostatin 1
Fig. 8. Schematic presentation of the study. Briefly, the <t>m6A</t> reader YTHDC2 is an endogenous ferroptosis inducer and its function is to suppress system XC −. In addition to what we’ve previously reported that the mRNA stability of SLC7A11 is suppressed by YTHDC2, in the present study we also found that SLC3A2, another subunit of system XC − can also be suppressed by YTHDC2 via targeting HOXA13-mediated transcription of SLC3A2, and the m6A methylation and subsequent destabilization of HOXA13 mRNA is a prerequisite for this process.
Ferrostatin 1, supplied by Tocris, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ferrostatin 1 fer 1  (Tocris)
93
Tocris ferrostatin 1 fer 1
Fig. 8. Schematic presentation of the study. Briefly, the <t>m6A</t> reader YTHDC2 is an endogenous ferroptosis inducer and its function is to suppress system XC −. In addition to what we’ve previously reported that the mRNA stability of SLC7A11 is suppressed by YTHDC2, in the present study we also found that SLC3A2, another subunit of system XC − can also be suppressed by YTHDC2 via targeting HOXA13-mediated transcription of SLC3A2, and the m6A methylation and subsequent destabilization of HOXA13 mRNA is a prerequisite for this process.
Ferrostatin 1 Fer 1, supplied by Tocris, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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etoposide merck 341205 ferrostatin 1  (Merck & Co)
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Merck & Co etoposide merck 341205 ferrostatin 1
Fig. 8. Schematic presentation of the study. Briefly, the <t>m6A</t> reader YTHDC2 is an endogenous ferroptosis inducer and its function is to suppress system XC −. In addition to what we’ve previously reported that the mRNA stability of SLC7A11 is suppressed by YTHDC2, in the present study we also found that SLC3A2, another subunit of system XC − can also be suppressed by YTHDC2 via targeting HOXA13-mediated transcription of SLC3A2, and the m6A methylation and subsequent destabilization of HOXA13 mRNA is a prerequisite for this process.
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hy-100579  (medchemexpress)
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medchemexpress hy-100579
Fig. 8. Schematic presentation of the study. Briefly, the <t>m6A</t> reader YTHDC2 is an endogenous ferroptosis inducer and its function is to suppress system XC −. In addition to what we’ve previously reported that the mRNA stability of SLC7A11 is suppressed by YTHDC2, in the present study we also found that SLC3A2, another subunit of system XC − can also be suppressed by YTHDC2 via targeting HOXA13-mediated transcription of SLC3A2, and the m6A methylation and subsequent destabilization of HOXA13 mRNA is a prerequisite for this process.
Hy 100579, supplied by medchemexpress, used in various techniques. Bioz Stars score: 98/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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antioxidant ferrostatin-1 fer-1  (Xcessbio Inc)
90
Xcessbio Inc antioxidant ferrostatin-1 fer-1
Fig. 8. Schematic presentation of the study. Briefly, the <t>m6A</t> reader YTHDC2 is an endogenous ferroptosis inducer and its function is to suppress system XC −. In addition to what we’ve previously reported that the mRNA stability of SLC7A11 is suppressed by YTHDC2, in the present study we also found that SLC3A2, another subunit of system XC − can also be suppressed by YTHDC2 via targeting HOXA13-mediated transcription of SLC3A2, and the m6A methylation and subsequent destabilization of HOXA13 mRNA is a prerequisite for this process.
Antioxidant Ferrostatin 1 Fer 1, supplied by Xcessbio Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ferrostatin-1 fer-1  (Topscience Co Ltd)
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Topscience Co Ltd ferrostatin-1 fer-1
Fig. 8. Schematic presentation of the study. Briefly, the <t>m6A</t> reader YTHDC2 is an endogenous ferroptosis inducer and its function is to suppress system XC −. In addition to what we’ve previously reported that the mRNA stability of SLC7A11 is suppressed by YTHDC2, in the present study we also found that SLC3A2, another subunit of system XC − can also be suppressed by YTHDC2 via targeting HOXA13-mediated transcription of SLC3A2, and the m6A methylation and subsequent destabilization of HOXA13 mRNA is a prerequisite for this process.
Ferrostatin 1 Fer 1, supplied by Topscience Co Ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ferrostatin-1 fer-1 sml0583  (Merck KGaA)
90
Merck KGaA ferrostatin-1 fer-1 sml0583
Fig. 8. Schematic presentation of the study. Briefly, the <t>m6A</t> reader YTHDC2 is an endogenous ferroptosis inducer and its function is to suppress system XC −. In addition to what we’ve previously reported that the mRNA stability of SLC7A11 is suppressed by YTHDC2, in the present study we also found that SLC3A2, another subunit of system XC − can also be suppressed by YTHDC2 via targeting HOXA13-mediated transcription of SLC3A2, and the m6A methylation and subsequent destabilization of HOXA13 mRNA is a prerequisite for this process.
Ferrostatin 1 Fer 1 Sml0583, supplied by Merck KGaA, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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ferroptosis inhibitor a4371  (ApexBio)
90
ApexBio ferroptosis inhibitor a4371
Fig. 8. Schematic presentation of the study. Briefly, the <t>m6A</t> reader YTHDC2 is an endogenous ferroptosis inducer and its function is to suppress system XC −. In addition to what we’ve previously reported that the mRNA stability of SLC7A11 is suppressed by YTHDC2, in the present study we also found that SLC3A2, another subunit of system XC − can also be suppressed by YTHDC2 via targeting HOXA13-mediated transcription of SLC3A2, and the m6A methylation and subsequent destabilization of HOXA13 mRNA is a prerequisite for this process.
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Image Search Results


Fig. 8. Schematic presentation of the study. Briefly, the m6A reader YTHDC2 is an endogenous ferroptosis inducer and its function is to suppress system XC −. In addition to what we’ve previously reported that the mRNA stability of SLC7A11 is suppressed by YTHDC2, in the present study we also found that SLC3A2, another subunit of system XC − can also be suppressed by YTHDC2 via targeting HOXA13-mediated transcription of SLC3A2, and the m6A methylation and subsequent destabilization of HOXA13 mRNA is a prerequisite for this process.

Journal: Free radical biology & medicine

Article Title: Targeting SLC3A2 subunit of system X C - is essential for m 6 A reader YTHDC2 to be an endogenous ferroptosis inducer in lung adenocarcinoma.

doi: 10.1016/j.freeradbiomed.2021.03.023

Figure Lengend Snippet: Fig. 8. Schematic presentation of the study. Briefly, the m6A reader YTHDC2 is an endogenous ferroptosis inducer and its function is to suppress system XC −. In addition to what we’ve previously reported that the mRNA stability of SLC7A11 is suppressed by YTHDC2, in the present study we also found that SLC3A2, another subunit of system XC − can also be suppressed by YTHDC2 via targeting HOXA13-mediated transcription of SLC3A2, and the m6A methylation and subsequent destabilization of HOXA13 mRNA is a prerequisite for this process.

Article Snippet: Abbreviations YTHDC2 YT521-B homology containing 2 LUAD lung adenocarcinoma SLC7A11 solute carrier 7A11 OS overall survival IB immunoblotting RT-qPCR reverse transcription-quantitative PCR IHC immunohistochemistry ELISA enzyme-linked immunosorbent assay Dox Doxycycline ROS reactive oxygen species EMSA electrophoretic mobility shift assay ChIP chromatin immunoprecipitation RIP RNA immunoprecipitation PAR-CLIP photoactivatable ribonucleotide crosslinking and immunoprecipitation SLC3A2 solute carrier 3A2 HOXA13 Homeo box A13 3′UTR 3′ untranslated region GSH glutathione GPX4 glutathione peroxidase RSL3 RAS-selective lethal 3 m6A N6-methyladenosine W writers E erasers R readers KP KrasG12D and p53R172H (HEK)-293T Human embryonic kidney BME Cultrex Basement Membrane Extract DFO Deferoxamine NAC N-acetyl-cysteine CHX cycloheximide 3-MA 3-Methyladenine ActD Actinomycin D Fer-1 Ferrostatin-1 DAA 3-Deazaadenosine Sh shRNA Sg single-guide RNA CST Cell Signaling Technology TMA tissue microarray assay 4-HNE 4-hydroxynonenal A adenosine C cytosine FISH Fluorescence in situ hybridization MDA malondialdehyde MeRIP-seq Methylated RNA Immunoprecipitation Sequening TSS transcription start site TF transcription factor POLR2L (poly II) RNA polymerase II, I and subunit L. L. Ma et al. Free Radical Biology and Medicine 168 (2021) 25–43

Techniques: Methylation